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THE RETINA INSTITUTE, New Orleans, LA, Premier Retinal Eye Care
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EPIRETINAL MEMBRANE

 

Different collagen types define two types of idiopathic epiretinal membranes

Michaela Kritzenberger  Benjamin Junglas  Carsten Framme  Horst Helbig  Veit‐Peter Gabel Rudolf Fuchshofer  Ernst R Tamm  Jost HillenkampFirst published: 12 April 2011 https://doi.org/10.1111/j.1365-2559.2011.03820.x Cited by: 20E R Tamm, Institute of Human Anatomy and Embryology, University of Regensburg, Universitätsstr. 31, D‐93053 Regensburg, Germany. e‐mail: ernst.tamm@vkl.uni‐regensburg.de SHARE

Abstract

Kritzenberger M, Junglas B, Framme C, Helbig H, Gabel V‐P, Fuchshofer R, Tamm E R & Hillenkamp J
(2011) Histopathology 58, 953–965

Different collagen types define two types of idiopathic epiretinal membranes

Aims:  To identify differences in extracellular matrix contents between idiopathic epiretinal membranes (IEM) of cellophane macular reflex (CMRM) or preretinal macular fibrosis (PMFM) type.

Methods and results:  Idiopathic epiretinal membranes were analysed by light and quantitative transmission electron microscopy, immunohistochemistry and Western blotting. Substantial differences between CMRM and PMFM were observed regarding the nature of extracellular fibrils. In CMRM the fibrils were thin, with diameters between 6 and 15 nm. Between the fibrils, aggregates of long‐spacing collagen were observed. In PMFM the diameters of fibrils measured either 18–26 or 36–56 nm. Using immunogold electron microscopy, 6–15 nm fibrils in CMRM were labelled for collagen type VI, while the fibrils in PMFM remained unstained. Using Western blotting and immunohistochemistry, a strong signal for collagen type VI was observed in all CMRM, while immunoreactivity was weak or absent in PMFM. In contrast, PMFM showed immunoreactivity for collagen types I and II, which was weak or absent in CMRM. Both types of membranes showed immunoreactivity for collagen types III and IV, laminin and fibronectin with similar intensity.

Conclusion:  The presence of high amounts of collagen type VI in CMRM and the relative absence of collagen types I and II is the major structural difference to PMFM.

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  • HOME
  • NEW PATIENTS
  • DR. EBRAHIM
  • REVIEWS
  • STORE
  • AMSLER GRID
  • VIDEO GALLERY
  • RESEARCH
  • GENETICS
  • ACUTE RETINAL NECROSIS
  • AUTOIMMUNE RETINOPATHY
  • BIRDSHOT CHORIORETINITIS
  • CANCER ASSOC. RETINOPATHY
  • CENTRAL SEROUS/POLYPOIDAL
  • COATS' DISEASE
  • CONE ROD DYSTROPHY
  • CYTOMEGALOVIRUS RETINITIS
  • DIABETIC RETINOPATHY
  • ENDOPHTHALMITIS
  • EPIRETINAL MEMBRANE
  • FLOATERS
  • HIGH BLOOD PRESSURE
  • HISTOPLASMOSIS
  • HIV RETINOPATHY
  • LATTICE DEGENERATION
  • MACULAR DEGENERATION
  • MACULAR HOLE
  • MELANOMA
  • METASTASES
  • MYELINATED RETINAL NFL
  • OCULAR ISCHEMIC SYNDROME
  • OPEN GLOBE INJURY
  • OPTIC NERVE HEAD DRUSEN
  • PROLIF. VITREORETINOPATHY
  • RETINAL ARTERY OCCLUSION
  • RETINAL DETACHMENT
  • RETINAL DYSTROPHIES
  • RETINAL VEIN OCCLUSION
  • RETINITIS PIGMENTOSA
  • RETINOSCHISIS
  • SARCOIDOSIS
  • SICKLE CELL RETINOPATHY
  • STARGARDT DISEASE
  • TOXOPLASMIC CHOROIDITIS
  • UVEITIS
  • VITREOUS DETACHMENT
  • VITREOUS HEMORRHAGE
  • PORTAL & PAYMENT